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Repression of 92-kDa type IV collagenase expression by MTA1 is mediated through direct interactions with the promoter via a mechanism, which is both dependent on and independent of histone deacetylation

  • University of Texas MD Anderson Cancer Center

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Pages (from-to)2309-2316
Number of pages8
JournalJournal of Biological Chemistry
Volume278
Issue number4
DOIs
StatePublished - Jan 24 2003
Externally publishedYes

ASJC Scopus Subject Areas

  • Biochemistry
  • Molecular Biology
  • Cell Biology

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